Lec 8 Teaching Prep: Part 3 — Chromatophore framing

2026-04-23 06:34:38 • 11:20

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Lecture 8 Teaching Prep Part 3.

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The New Chromatto IV Framing Block.

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Hey Michael, this is Part 3 of your prep for Lecture 8, and it is a focus briefing on

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a block of material you have just added to the top of the pigment section.

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Part 1 was your neural processing prep.

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Part 2 was the pigment's block as originally designed.

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This Part 3 covers the four new slides that now sit between the Block 2 title card and

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the what is a pigment slide, and it explains what those slides are doing, how they should

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land in the room, and how they change the rhythm of the pigment's block.

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Listen to this on the commuting.

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It is about 10 minutes long, and it is the last thing you need before you walk in.

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Let's start with the big idea.

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Your original pigment's block jumped directly from the Block 2 title to the Chromat 4 and

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conjugation chemistry.

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That was efficient, but it skipped a level.

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This needed to know where in the animal's body color lives, and they needed a conceptual

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anchor for the fact that some animal colors are not pigments at all.

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The new four slide opener fixes that.

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It goes cell, then mechanism, then a short detour on white, then evolution.

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Only after all of that do you reach the chromophore slide.

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Here is the arc slide one, where does color live in an animal body?

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Slide 2.

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Chromatto IVs use two mechanisms, pigments or structure.

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Slide 3.

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Does a white pigment actually exist?

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Slide 4.

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Why mammals only have melanocytes?

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The nocturnal bottleneck again.

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Total added time.

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About 7 minutes.

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To absorb it, compress the opponent's ETSD brief at the start of Block 2 and the MC1 are

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convergence examples in Block 2.

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One minute off each gets you close.

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Now the substance of each new slide.

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Slide 1 is the cellular framing.

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The core message is this.

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In animals, color is produced by specialized cells called chromatophores and invertebrates

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every single one of those cells traces to the neural crust.

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You do not need to teach neural crust development in depth.

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You just need students to hold onto two facts.

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First, color in a vertebrate is not chemistry painted onto the skin.

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It is the output of a developmental cell lineage that migrates during embryogenesis.

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Second, the vertebrate starter kit includes six distinct chromatophore types of which three

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are pigment-based and two are structural and one is a weird outlier.

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The table on the right of the slide does the work of introducing all six.

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Do not read every row.

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Point to it.

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Say three of these are pigment cells, two are structural cells.

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One is a blue pigment oddity found mainly in mandra fish.

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Then move on.

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A good verbal line for this slide, color in a zebra is not zebra pigment.

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It is melanocytes where they should be, plus a spatial developmental program telling

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them when to turn on.

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That line sticks and previews the pattern placement theme you hit later with MC1R and zebra

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stripes.

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One trap.

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Do not get pulled into a tangent on neural crust derivatives.

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If a student asks, you can say, yes, the neural crust is sometimes called the fourth germ

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layer.

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It also produces shwan cells, peripheral neurons, jaw cartilage and much of the skull.

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Come to office hours and we can talk about why evolutionary biologists find that so interesting.

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Then advance.

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Slide two is the conceptual pivot.

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Chromatophores use two mechanisms, pigment absorption or structural scattering and interference.

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Two columns side by side.

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Structural based chromatophores, melanophores, xanthophores, erythrophores contain colored

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molecules whose chromophores absorb specific wavelengths.

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The color lives in the molecules electrons.

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Structural chromatophores, iridophores and lycophores contain no colored pigment at all.

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The color lives in the cell's architecture.

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You are going to want to linger on iridophores for about 15 seconds because students will

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not have encountered them before.

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The line that works, iridophores are filled with stacks of guanine crystals alternating

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with layers of cytoplasm.

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Light entering that stack reflects off every interface.

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When the layer thicknesses are tuned to visible wavelengths, you get constructive interference.

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Same physics as a soak bubble, a peacock feather or an oil slick.

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That is how a goldfish gets its silver belly.

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That is how a chameleon changes color by actively spacing the crystals closer or further

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apart.

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Look-a-fores get one sentence, dense purine granules, broadband scattering, diffuse white,

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the waste snow and milk are white.

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You do not need to do more than that.

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Slide 3 will come back to look-a-fores.

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The punchline for slide 2 is the line at the bottom, same embryological lineage, two completely

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different physics.

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Say that explicitly.

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Then make the pointer to Lex 9 and 10.

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Tell them, we focus on the pigment side today because it is the simpler story.

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The structural side, where most of the spectacular blues and greens in nature come from, is

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LEC 9 and 10.

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Keep that pointer in your head as we move through the pigment families.

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Slide 3 is that does a white pigment actually exist a side.

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This is the slide that fixes a real conceptual sloppiness that used to haunt the tern section.

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The original tern slide said, look-a-pterine produces white.

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That is the shorthand, but it is misleading because LEC 9 itself absorbs only in the

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UV and is colorless to our eyes.

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The white cabbage wing color is a scattering phenomenon, not an absorption phenomenon.

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You need 90 seconds on this slide.

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Not more.

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Just walk through the three mechanisms.

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First, scattering crystals, LEC 9 in pirates, guanine and fish look-a-fores.

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The pigment absorbs in the UV.

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The white is physical scattering.

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Second, absence of a working pigment, zebra white stripes, pie-balled mammals, fiddle

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ligo, albino animals.

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The melanocytes are missing or silenced or broken.

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No chromatic absorber, so the tissue scatters broadly.

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Third, scattering keratin, polar bear fur with its hollow air-filled medulla, air pockets

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inside feather barbs.

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Physical scattering structures, not pigments.

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The rule you want in their notebooks, white in animals is either a missing pigment or

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a scattering structure.

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Never a molecule that absorbs every color of white.

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Tell them explicitly that this matters for the tarant slide later in the lecture.

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You will remind them when you get there.

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Slide 4 is the payoff slide.

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Why mammals only have melanocytes?

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The nocturnal bottleneck again.

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This is the second time you have invoked the nocturnal bottleneck in this course.

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The first time was for opts and loss and mammalian dichromacy in the photoreceptor evolution

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lecture.

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Today you are using it to explain the chromatophore complement.

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Point that out.

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Say, the same evolutionary episode that shaped what mammals can see also shaped what mammals

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can look like.

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Both the input and the output of the color system passed through the same bottleneck.

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That is the line.

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The comparative table on the right does the work.

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Fishes, five or six chromatophore types.

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Amphibians, three.

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Reptiles, three or four.

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Birds, one chromatophore type, just the melanocyte, but they reinvented structural color using

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keratin nanostructures and feather barbs.

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Mammals, one.

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Just melanocytes.

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Full stop.

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The two reasons mammals lost the others.

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Relax selection during the nocturnal bottleneck, plus forgetting in the way of iridophore signaling

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anyway.

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Both matter.

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Primary cause is neural crest fate loss during the bottleneck.

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Secondary is the fur compatibility issue.

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The line that really lands in the room.

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That is why your cat is always a variation on black, brown, or orange, never-teal, silver,

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or iridescent blue.

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Students love that line.

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It caches out the whole story in a concrete image they can picture instantly.

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Deliver it, pause, advance.

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First slide four, you hit though what is a pigment, chromophore slide, and from there

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you are back to the original part to script.

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Everything from the chromophore slide onward is unchanged.

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A few things to watch for.

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First, do not let the four intro slides brawl.

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They are supposed to be framing, not a deep dive.

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Seven minutes total.

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If you find yourself on slide one for more than two minutes, advance.

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If you find yourself explaining thin film interference mechanics in detail on slide two,

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stop and say physics next week and advance.

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The students do not need to understand I writ of four optics today.

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They just need to know I writ of fours exist and are not pigment cells.

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Second, the white pigment slide is an aside.

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Do not build it up as a major concept.

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Walk through the three mechanisms quickly, state the rule, and move on.

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The reason you want to flag it now is purely so that when students here look up during

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white 15 minutes later, they think scattering instead of white pigment chromophore.

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That is the only job that slide needs to do.

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Third, the nocturnal bottleneck callback is the emotional peak of the new block.

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Give it the full delivery.

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The connection between obs and loss and chromatophor loss, as two outputs of the same historical event,

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is exactly the kind of synthesis students remember.

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This is also where you get to reward students who have been paying attention all quarter.

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They know what the nocturnal bottleneck is.

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They know mammals are mostly dichromatic, and now you are using those prior facts as building

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blocks.

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Slow down.

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Land it.

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Fourth, when you finally get to the chromophore slide after the four new introslides, you

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may feel the energy dip.

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It's a lot of setup.

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Pick up the pace.

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The chromophore slide is a conceptually dense slide that deserves the full four minutes

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of original material, the spring analogy, the conjugation length axis, the demo three

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callback.

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Do not skip any of that to recover time.

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Recover time later, in the MC1 are examples and the Opponancy TPSD brief.

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One more timing note.

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The Terran Gallery slide now has a new click reveal where you give the white pigment clarification

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a second time.

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Keep it short 15 seconds.

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Just say, remember the rule from the opening framing.

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Lucoptor and absorbs in the UV.

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The wing is white because the crystal scatter.

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White is a missing pigment or a scattering structure, never a chromophore.

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Then move to porphorins.

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Big picture.

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The new framing block does three things at once.

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It gives students a cellular anchor so the rest of the pigment chemistry has somewhere to

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sit.

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Slags structural color as a mechanism so the mystery of blue slide later in the lecture

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lands cleanly.

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And it gives you another return to the nocturnal bottleneck narrative that has been running

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through the whole course.

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The cost is seven minutes.

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The payoff is a much more coherent block too.

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You have this.

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Trust the architecture.

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Trust the students.

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Trust your instinct to move when you are tempted to over explain.

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Have a great lecture.